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mouse creatine kinase mb elisa kit  (Novus Biologicals)
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Novus Biologicals mouse creatine kinase mb elisa kit
Figure 1. PHB2 overexpression attenuates LPS-induced myocardial depression. WT mice and PHB2 transgenic (PHB2Tg) mice were administered LPS to induce endotoxemia-related myocardial dysfunction. (A-F) Analysis of cardiac function via echocardiography. (G-I) Serum levels of TnT, CK-MB, <t>and</t> <t>BNP</t> were determined by <t>ELISA.</t> (J) Histopathological evaluation of cardiac tissue (HE staining). (K) Electron microscopy (EM) was applied to observe ultrastructural changes in myocardium. Cardiomyocyte swelling, fragmented cardiomyocyte nuclei, and twisted myocardial fibers are marked with red arrows. (L, M) RT-qPCR analysis of the transcription of CXCR7 and TNFα in cardiac tissue. #p<0.05 vs. PBS+WT group; *p<0.05 vs. LPS+WT group.
Mouse Creatine Kinase Mb Elisa Kit, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Figure 1. PHB2 overexpression attenuates LPS-induced myocardial depression. WT mice and PHB2 transgenic (PHB2Tg) mice were administered LPS to induce endotoxemia-related myocardial dysfunction. (A-F) Analysis of cardiac function via echocardiography. (G-I) Serum levels of TnT, CK-MB, and BNP were determined by ELISA. (J) Histopathological evaluation of cardiac tissue (HE staining). (K) Electron microscopy (EM) was applied to observe ultrastructural changes in myocardium. Cardiomyocyte swelling, fragmented cardiomyocyte nuclei, and twisted myocardial fibers are marked with red arrows. (L, M) RT-qPCR analysis of the transcription of CXCR7 and TNFα in cardiac tissue. #p<0.05 vs. PBS+WT group; *p<0.05 vs. LPS+WT group.

Journal: International journal of biological sciences

Article Title: Pgam5-mediated PHB2 dephosphorylation contributes to endotoxemia-induced myocardial dysfunction by inhibiting mitophagy and the mitochondrial unfolded protein response.

doi: 10.7150/ijbs.85767

Figure Lengend Snippet: Figure 1. PHB2 overexpression attenuates LPS-induced myocardial depression. WT mice and PHB2 transgenic (PHB2Tg) mice were administered LPS to induce endotoxemia-related myocardial dysfunction. (A-F) Analysis of cardiac function via echocardiography. (G-I) Serum levels of TnT, CK-MB, and BNP were determined by ELISA. (J) Histopathological evaluation of cardiac tissue (HE staining). (K) Electron microscopy (EM) was applied to observe ultrastructural changes in myocardium. Cardiomyocyte swelling, fragmented cardiomyocyte nuclei, and twisted myocardial fibers are marked with red arrows. (L, M) RT-qPCR analysis of the transcription of CXCR7 and TNFα in cardiac tissue. #p<0.05 vs. PBS+WT group; *p<0.05 vs. LPS+WT group.

Article Snippet: 19 https://www.ijbs.com 4660 Mouse TNT/Troponin T ELISA Kit (F55154, LifeSpan BioSciences), Mouse Brain Natriuretic Peptide (BNP) ELISA Kit (CSB-E07971m, CUSABIO), and Mouse Creatine Kinase MB ELISA Kit (NBP2-74312, Novus Biologicals) were used to determine the activity/ concentration of Bax, Bcl-2, Gpx4, Ptx3, Txnrd2, BNP, TnT, and CK-MB [47].

Techniques: Over Expression, Transgenic Assay, Enzyme-linked Immunosorbent Assay, Staining, Electron Microscopy, Quantitative RT-PCR

Figure 7. Pgam5 deletion improves myocardial function in LPS-treated mice. WT mice and cardiomyocyte-specific Pgam5 knockout (Pgam5Cko) mice were administered LPS to induce endotoxemia-related myocardial dysfunction. (A-F) Analysis of cardiac function via echocardiography. (G-I) Serum levels of TnT, CK-MB, and BNP were determined by ELISA. (J) Cardiac histopathology analysis by HE staining. (K) Electron microscopy (EM) was applied to analyze ultrastructural changes in myocardium. Cardiomyocyte swelling, fragmented cardiomyocyte nuclei, and twisted myocardial fibers are marked with red arrows. (L, M) RT-qPCR analysis of CXCR7 and TNFα mRNA levels in heart tissues. #p<0.05 vs. PBS+Pgam5Cko group; *p<0.05 vs. LPS+Pgam5Cko group.

Journal: International journal of biological sciences

Article Title: Pgam5-mediated PHB2 dephosphorylation contributes to endotoxemia-induced myocardial dysfunction by inhibiting mitophagy and the mitochondrial unfolded protein response.

doi: 10.7150/ijbs.85767

Figure Lengend Snippet: Figure 7. Pgam5 deletion improves myocardial function in LPS-treated mice. WT mice and cardiomyocyte-specific Pgam5 knockout (Pgam5Cko) mice were administered LPS to induce endotoxemia-related myocardial dysfunction. (A-F) Analysis of cardiac function via echocardiography. (G-I) Serum levels of TnT, CK-MB, and BNP were determined by ELISA. (J) Cardiac histopathology analysis by HE staining. (K) Electron microscopy (EM) was applied to analyze ultrastructural changes in myocardium. Cardiomyocyte swelling, fragmented cardiomyocyte nuclei, and twisted myocardial fibers are marked with red arrows. (L, M) RT-qPCR analysis of CXCR7 and TNFα mRNA levels in heart tissues. #p<0.05 vs. PBS+Pgam5Cko group; *p<0.05 vs. LPS+Pgam5Cko group.

Article Snippet: 19 https://www.ijbs.com 4660 Mouse TNT/Troponin T ELISA Kit (F55154, LifeSpan BioSciences), Mouse Brain Natriuretic Peptide (BNP) ELISA Kit (CSB-E07971m, CUSABIO), and Mouse Creatine Kinase MB ELISA Kit (NBP2-74312, Novus Biologicals) were used to determine the activity/ concentration of Bax, Bcl-2, Gpx4, Ptx3, Txnrd2, BNP, TnT, and CK-MB [47].

Techniques: Knock-Out, Enzyme-linked Immunosorbent Assay, Histopathology, Staining, Electron Microscopy, Quantitative RT-PCR